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Moreover, animal and translational studies that suggest plausible neurodevelopmental effects at doses close to the therapeutic range were briefly mentioned in relation to biological plausibility, whereas recent experimental literature points to specific mechanistic hypotheses (e.g., CB1 modulation, OS, sexspecific prefrontal transcriptomic changes, epigenetic programming) deserve more neutral discussion (Philippot et al., 2018[86])

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However, clinical evidence and UK regulatory guidance confirm that these medications can cause sexual side effects, including erectile dysfunction, in a subset of users

Moreover, hyper-succinylation of nuclear factor erythroid 2-related factor 2 (NRF2) mediates a multi-drug-resistant (MDR) phenotype by upregulating resistance protein MRP1 and other drug-inactivating enzymes [121]