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The deficit in mitochondrial handling of methylmalonyl-CoA that is common to the mut, cblA, and cblB forms of methylmalonic acidemia can be demonstrated in vivo by administration of the precursor, [1-13C]propionate, with measurement of 13CO2 in expired air (11)

Moreover, to further characterize the mechanism of action of caffeine on alcohol-mediated stimulation of mesolimbic DA neurons, in-vitro patch-clamp recordings from pVTA slices were performed

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