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Unlike apoptosis, necroptosis does not rely on caspase activity, but rather is mediated through the cascade activation of key molecules such as RIPK1, RIPK3, and MLKL, which triggers cell membrane rupture and release of cellular contents, thereby activating the immune system [251]

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Notably, SFN + EXO treatment effectively normalized Nrf2 expression to levels comparable with healthy controls and provided sufficient antioxidant defense to mitigate ROS without inducing Nrf2 overactivation