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The increase in the production of intracellular ROS is due to various mechanisms including the intensification of ATP production to support the rise in cellular components, alterations in the signaling pathways associated with cancer metastasis, and to the hyper activation of several oncogenes [115]

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Low level phosphorylation of histone H2AX on serine 139 (H2AX) is not associated with DNA double-strand breaks

For instance, while oncogenic KRAS mutations increase cellular oxidative stress and sensitize cells to ferroptosis, they concurrently upregulate FSP1 as a key resistance mechanism [77]

Pharmacokinetic and pharmacodynamic considerations for NMDA-receptor antagonist ketamine in the treatment of chronic neuropathic pain: an update of the most recent literature

Lack of it often results in denied claims