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The K D of NBD-PC binding to NPC2 is more than 100-fold smaller when the lipid concentration is below the CMC compared with above, but different photophysical mechanisms underlying the binding-induced fluorescence increase of NBD-lipids prevent a quantitative comparison of K D -values below and above their CMC (28)

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Its probable mechanism includes activation of renal AGE-RAGE signaling pathways, decreasing levels of monocyte chemotactic protein-1 (MCP-1) and PKC-A, which consequently reduces the production of slit diaphragm proteins such as nephrin and podocin, thereby mitigating the impact of AGE-mediated DN pathogenesis (Muthenna et al., 2014)