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This shift results in abnormal sodium and calcium influx, rendering the axons hyperexcitable and generating spontaneous ectopic discharges that act as a persistent false signal of ongoing tissue damage
Toxic Subst Mech 1999
M., Walther, J
Oxidative damage and metabolic dysfunction in Huntingtons disease: Selective vulnerability of the basal ganglia
This unique characteristic of GSTLs help in the substitution of amino acid in active site residues forming mixed disulfides with thiol group rather than promoting formation of reactive thiolate anion of GSH
Furukawa, A., Tada-Oikawa, S., Kawanishi, S
