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Journal of Neurochemistry, 97 (2), 303309

Chang, H., Tsai, C., Hsu, H., & Pang, H

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Conclusions and Prospects Taken together, gliotoxin can show cytotoxic effects via antiangiogenesis, inflammation, immunosuppression mediated by the NF-B and DNA damage, peroxide damage mediated by production of ROS mediated by the inhibition of NADPH oxidase and redox cycling, and apoptosis via various signal pathways, including caspase-dependent mitochondrial membrane potential disruption, the NF-B pathway and NOTCH 2 -dependent apoptosis