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Aims/hypothesis Barth syndrome is a mitochondrial disorder caused by Tafazzin (TAZ) mutations, which impair cardiolipin remodelling and contribute to systemic metabolic alterations

It is involved in many biological processes including detoxification of xenobiotics, removal of oxygen-reactive species, regulation of the redox state of cells and the oxidative state of important protein sulfhydryl groups, and regulation of immune function

All participants underwent a 5-day modified fasting therapy introduced with 2-day moderate calorie restriction

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AOD9604 10MG Modifiziertes Wachstumshormon-Fragment AOD9604 ist ein synthetisches Fragment des menschlichen Wa