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While prompt and efficient healing of skin wounds is essential to maintaining the vital barrier function of the skin, a pathological wound healing response can result in abnormal extracellular matrix (ECM) deposition and diminished degradation, ultimately leading to scarring and fibrosis (Figure 2)

Therapeutic strategies to activate the TST pathway are explored, with a focus on sodium thiosulfate (STS), novel small molecule (Hit 2), and recombinant hTST protein

doi: 10.1016/j.freeradbiomed.2021.10.027

3 These features have inextricable links to the orchestration of redox signaling, which enables DTP cancer cells to survive constant stress over the course of treatment by developing adaptive mechanisms to avoid endogenous or exogenous oxidative stress (Fig

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Barriers to Linkage to Care for Hepatitis B Virus Infection: A Qualitative Analysis in Burkina Faso, West Africa
