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However, growing evidence indicates that alternative metabolic routes such as malic enzyme 1 (ME1), isocitrate dehydrogenases (IDH1 and IDH2), and the folate cycle also significantly contribute to NADPH production, particularly under stress conditions or during metabolic reprogramming [43]

However, a study carried out by Abdel-Wahhab et al

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Chemical Information: BPC-157

Platelet-rich plasma activates proinflammatory signaling pathways and induces oxidative stress in tendon fibroblasts