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Decreased acetylation of H4 occurred at 1200 and 900 bp in the NC group, at 1200 bp in the CC group, and at 1500 and 900 bp in the SC group

GSH depletion is necessary for the formation of the apoptosome [124] and also triggers cell death by modulation of the permeability transition pore of the mitochondria and the activation of executioner caspases [125,126]

26 Given the burgeoning interest in copper metabolism and cuproptosis, it is crucial to develop a comprehensive understanding of the molecular mechanism and therapeutic landscape of copper and cuproptosis in cancer

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ROS trigger apoptosis by activating the pro-apoptotic proteins caspase-3/7/9 and cleaved PARP, disrupting mitochondrial function, and in pancreatic cancer melatonin-induced ROS enhance apoptosis through the mitochondrial pathway (Florido et al., 2022b)