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doi: 10.1159/000465981
Targeting other KRAS mutations and indirect strategies Targeting other KRAS mutations The successful development of KRAS (G12C) inhibitors is dependent on the covalent inhibition of cysteine and high GTPase activity of KRAS (G12C), which is not fully present in other KRAS-mutant types 114
Chen Yao, [email protected]
PMID: 42174621 Biomed PharmacotherTargeting ER stress in adrenocortical carcinoma: Celastrol as a novel therapeutic candidate
2007, Venkatesh et al
Changes in membrane currents in bullfrog atrium produced by acetylcholine
