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Acknowledgements The figures were created using images from the Icon Library on www.biorender.com

[DOI] [PMC free article] [PubMed] [Google Scholar] 233.Buglio D.S., Marton L.T., Laurindo L.F., Guiguer E.L., Arajo A.C., Buchaim R.L., Goulart R.A., Rubira C.J., Barbalho S.M

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Specifically, we identified KU-57788 as a DRP1 agonist that induces excessive mitochondrial fission, which in turn activates the NRF2/SLC7A11/GSH axis as a protective response in ATC cells to mitigate its cytotoxic effects

corrected the Q456X and I368N mutations of PINK1 in iPS cells from a patient with PD, which improved cellular metabolism, enhanced the DA neuron differentiation potential and reduced astrocyte enrichment, indicating that restoring the PINK1 PRKN axis normalizes key disease-relevant phenotypes 40

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