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Description
Specifically, the data implied that colonic epithelial inflammation was the result of a metabolic abnormality, which resulted in impaired redox homeostasis leading to excess production of hydrogen peroxide by colonic epithelial cells
P max : maximal power
Regulation of the structure and activity of pyruvate carboxylase by acetyl CoA
Leucine activates mTOR pathways, which tells cells to grow and to make new proteins
Exhaustion of CD8 + T cells impairs tumor immunity and contributes to the uncontrolled growth of the tumor [85]
Gpx4 at the crossroads of lipid homeostasis and ferroptosis
