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On cellular metabolic demand, mTORC1 and mTORC2 initiate biosynthetic cascades to support cell proliferation and anabolic state ( FIGURE 3 mTORC1 promotes protein synthesis by phosphorylating eukaryotic initiation factor 4E-binding proteins (4E-BPs) and p70 S6 kinase 1 (S6K1), increasing the production of ATP, nucleotides, and lipids

we would recommend several excellent reviews on this topic (Cadet et al., 2013

Hubner S, Konopleva M, Zeng Z, Hoff FW, Qiu Y, Ruvolo V, Horton TM, Kornblau SM

Safety concerns, including hypokalemia, sedation, and teratogenicity, also restrict use

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Subsequently, they conducted in vivo experiments to validate the tumor-suppressive role of MIEAP and found that the concurrent knockout of MIEAP and autophagy-related gene 5 (ATG5) accelerated TC development and induced the oncocytic phenotype in BRAF V600E -positive thyroid cells [169]