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For example, in primary cardiac cells, Cu 2+ increases the release of interleukin-6 (IL-6) and activates MAP kinases, which are linked to cardiac inflammation and hypertrophy [76]

Within synaptic vesicles, amphetamine disrupts vesicular monoamine storage through two complementary mechanisms: (A) diffusion into vesicles and proton buffering, which collapses the vesicular proton gradient required for vesicular monoamine transporter 2 (VMAT2) function