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Mutations in ENPP1 are associated with idiopathic infantile arterial calcification

The restoration of this single glycine residue is thus sufficient to recover the GSH efflux stimulation, but not the drug transport, which further suggests a different mechanism for these two transport activities

a , Total GPX4 activity of HT1080 cells overexpressing GFP-GPX4 WT , GFP-GPX4 K48A , or GFP-GPX4 K48L and a control line

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They can also strengthen the mucosal barrier [80] and reduce intestinal permeability, upregulating TJS proteins [81]

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