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Firstly, the main strength is the long study follow-up (15 months), which allows for gathering information about potential changes in patients QoL in response to long-term ALC treatment, usually not be easily detectable in studies with shorter observation periods

In the most severe cases, this overreaction leads to a cytokine storm, wherein various proinflammatory cytokines are produced at a much higher rate than normal

Furthermore, significant alterations in the carnitine/acylcarnitine pathway were detected in bladder cancer patients

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Therefore, mitochondria-associated synaptic abnormalities are likely to contain robust therapeutic targets for ASD that have yet to be discovered