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73, 74, 75, 76 Taken together, an acceptable hypothesis appears to be that increased claudin-2 expression in inflamed mucosa of IBD patients may help promote epithelial injury and repair

Some research suggests the peptide may enhance mitochondrial fatty acid oxidation through AMPK activation or other metabolic signaling pathways, though this mechanism requires further validation

You start, we follow up, we adjust as needed

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The use of the patients own fat helps ensure that the augmentation is well-integrated into the body

Published data spans multiple GI research contexts: gastric ulcer models, inflammatory bowel disease research, esophageal injury, and intestinal anastomotic healing
