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Zhang H, Go YM, Jones DP (2007) Mitochondrial thioredoxin-2/peroxiredoxin-3 system functions in parallel with mitochondrial GSH system in protection against oxidative stress

They also uncovered a homeostatic mechanism that Ca2+ depletion can trigger influx of cytosol GSH into ER

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The excretion rate of transferrin in NZB/W mice significantly increased with the production of urinary protein, indicating increased tubular exposure and potential tubular reabsorption

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Patients were also required to have no prior cytotoxic chemotherapy or immunotherapy (except steroids) and have an ECOG performance status of 0 or 1 (KPS70%), ANC 1.5x10 9 /L, platelets 100x10 9 /L, serum creatinine 1.5 ULN, total bilirubin 1.5 ULN, AST/ALT 2.5 ULN and CEA within normal limits at time of randomization