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Li Y, Soto-Ramirez ZN, Roscher J, Medvec T, Alaoui-El-Azher M, Piazza P, et al
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Together, increased intraplaque cellular death and dysfunctional efferocytosis as a result of G6PD-d may accelerate plaque growth, necrotic core formation and the development of an inflammatory plaque microenvironment 106,107 (Fig
by increasing stomach acidity or liver enzyme elevations)
Moreover, the surface modification of the NPs with targeting ligands can further improve the specificity and efficacy of BDNF delivery, which promotes the survival and function of the neurons (183)
Insulins direct hepatic effect explains the inhibition of glucose production caused by insulin secretion
