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The best studied one is the increase of the VEGF pathway (Vascular Endothelial Growth Factor), indicating the production of new blood vessels (angiogenesis)

Accumulation of misfolded proteins that lead to ER stress triggers the UPR via activation of three signaling arms coordinated by IRE1-XBP1, PERK-eIF2a-ATF4, and ATF6 [78]

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establishment of longitudinal follow-up systems capable of assessing predictive efficacy beyond mere diagnostic discrimination