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Nicotinamide N-methyltransferase: an emerging protagonist in cancer macro(r)evolution

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Although endothelial activation and redox signaling compose a normal host defense, when this activation is sustained, chronic production of ROS may exceed the capacity of cellular enzymatic and nonenzymatic antioxidant defenses [49], contributing to triggering vascular diseases such as atherosclerosis or peripheral artery disease [102, 103]

Several papers discuss the compound in relation to angiogenesis, nitric oxide systems and endothelial response