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However, the detected deficits are in good agreement with the here observed hippocampal hypometabolism and synaptic alterations in the hippocampus of Tg4-42 mice 22

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H.MatsubaraH.ImaiT.YoshiokaY.KatsukiJ.et al (2023)
Bandgar, Prajakta S
No inhibition of the phosphorylation of p38 mitogen-activated protein (MAP) kinase, c-Jun N-terminal kinase (JNK), or extracellular signal-regulated kinase (ERK) was observed as a result of NAC-S2 treatment, suggesting that supersulfides specifically target NF-B signaling [77]
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