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Animal studies suggest that high concentrations of 1,25(OH)2D are necessary to generate Tregs when supplemented, but this can lead to adverse side effects such as hypercalcemia

Selective induction of endogenous NQO1 was seen in metabolically relevant target tissues of Keap1 -KO mice including skeletal muscle, liver, and WAT while expression of other Nrf2 targets such as the antioxidants GCLC, HO-1 and SOD2 was minimally impacted

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The current focus on regulating the ferroptosis signaling pathway for depression treatment is primarily limited to animal models, lacking clinical research to validate its efficacy in patients

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