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First, EGCG can reduce mitochondrial ROS, then to further prevent mitochondrial respiratory enzymes injury

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The anti-atherosclerotic effects of SIN (SIN) are attributed to its ability to inhibit inflammation progression, modulate immune cell functions, and suppress smooth muscle cell proliferation

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This also supported our hypothesis that the deletion of gsh-px enhanced the adhesion and invasion ability of L

The outer MSN layer provides immobilization sites for chemically conjugating MGF, a variant subtype of IGF-1, via EDC/NHS chemistry