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While anecdotal evidence exists, rigorous clinical trials are lacking

To investigate whether GCG, VGF, SCG2, SCG3, and CHGA contribute to hyperglucagonemia in individuals with diabetes and diabetic mice, we examined gene expression data from cells in patients with type 2 diabetes (GSE84133) 42 and high-fat diet (HFD)-induced obese mice (GSE203151) 43

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This connection bridges the GCH1-BH4-DHFR pathway and the FSP1-CoQ10 axis, whose collaboration impedes the progression of ferroptosis