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Our recently conducted preclinical studies in the penetrating TBI animal model, demonstrated zero toxic effects for IP-administered NACA at the highest concentration (600 mg/kg), supporting a safer profile for its use as a mitochondrial-targeted neuroprotection compound for TBI (unpublished data)
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Under the optimized experimental conditions, keeping the acquisition method and average times constant, the enhancement factor was calculated to be 128 with the intensity at the characteristic absorption peak of 1,654 cm 1
AMPT exerts its inhibitory actions on tyrosine hydroxylase, which will lead to a severe impairment in the endogenous production of dopamine [25, 31]
10 - Preclinical models for wound-healing studies
