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Elevated intracellular iron can saturate NMs binding sites, causing iron release from low-affinity sites and enhancing iron-driven oxidative stress (128)

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This polymorphism, which is also inherited as an autosomal recessive trait, occurs in 35% of Caucasians and 1823% of Japanese populations

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200,201 Moreover, the methionine metabolism catalyzed by NNMT diverts the NAM from the NAD + salvage pathway

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