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This process leads to reduced glucose absorption in tissues and heightened insulin secretion by pancreatic -cells [77]

Certain enteric pathogens possess mechanisms that enable their direct adherence to IECS while others permit the secretion of effector proteins or enterotoxins via membrane localised secretion systems that typically target TJs thereby increasing intestinal permeability by disrupting the intracellular cytoskeleton of IECs (Knig et al

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Initial treatment with activated charcoal and phosphatidylcholine resulted in mild improvement and partial reduction in inflammatory markers