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Investigators have proposed that GHK-Cu's central function is to deliver copper to fibroblasts in a controlled, non-toxic form buffering against the redox damage that free Cu would otherwise cause [4]

Pharmaceutical Research (1999) 7

Springer, US, New York, NY, pp 2127 Hiller F, Besselt K, Deubel S et al (2015) GPx2 induction is mediated through STAT transcription factors during acute colitis

in maintenance you inject about every other active day (e.g., Mon/Wed/Fri)

The transcriptional repressor Nab1 is a specific regulator of pathological cardiac hypertrophy

Because NO regulates vasodilation, endothelial survival, and angiogenic signaling, this balancing may support perfusion of injured cellular structures and facilitate endothelial activation and vessel remodeling during repair