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The latter is chemically unstable due to the reactive carbon of the isothiocyanate group (N=C=S) that easily reacts with nucleophiles such as thiols

Conversely, with conditions leading to neurodegeneration or chronic inflammation, oxidative stress persists long enough to lead to abnormal immune response pathwaysparticularly NF-kband prolonged treatment with GSH may lead to reduced redox tone and thus attenuating NF-kB, causing an anti-inflammatory response instead of an immuno-supportive one (Limongi et al., 2019

Abstract The plasma membrane protein caveolin-1 (CAV-1) regulates signaling by inhibiting a wide range of kinases and other enzymes

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Glutathione-bound Mercury(II) triggers the catalytic depletion of the cellular GSH pool by a catalytic mechanism, which involves the presence of Mercury as the glutathione conjugate

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