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NF-B is a key regulator of apoptosis, immunity, aging, and inflammation due to its role in the expression of proinflammatory genes, including cytokines, chemokines, and adhesion molecules.34 In the present study, the absence of elevated NF-B, TNF-, and IL-6 levels in melatonin- and glutathione-treated groups suggests potential modulation of inflammatory responses by these agents

doi: 10.1097/MCO.0b013e32835ad2ee 18 BertoniG.TrevisiE.HanX.BionazM

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10.1007/s00535-004-1469-1468 J

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1,2,6 The resultant abnormal erythropoiesis can trigger other notable abnormal laboratory findings, such as decreased haptoglobin levels, high lactate dehydrogenase levels, and elevated reticulocyte count