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The increased activity of ERK1/2 led to an increase in the proteasome chymotrypsin-like activity, whereas the increased activity of MEK1 resulted in a lower proteasome chymotrypsin-like activity

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[DOI] [PMC free article] [PubMed] [Google Scholar] Hanigan M.H., Devarajan P

FAK-paxillin signaling and F-actin cytoskeletal organization Molecular Modulation of the NO-system, VEGFR2 signaling, growth-hormone receptor expression in fibroblasts, and interactions with dopamine- and serotonin-related neurotransmitter pathways Systemic (Preclinical) Rodent models of gastrointestinal mucosal lesions, liver injury, tendon and ligament transection, and vascular occlusion syndromes References (BPC-157): Sikiric P, et al