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In rats, the KA-SE model also exhibits a decrease in reduced glutathione (GSH) and an increase in its oxidized form (GSSG), indicating increased ROS burden post seizure activity

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An increase in the activity of the Wnt/-catenin signaling pathway (5)

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Oxidative stress has been reported in kidney disease due to both antioxidant depletion and increased ROS production, which can accelerate kidney disease progression [46]