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I., Jurisicova A., et al

Conversely, downregulation of CHAC1/BOTCH was associated with increased neuronal damage and impaired recovery ( The regulation of CHAC1/BOTCH involves additional molecular players: during intracerebral hemorrhage, inhibition of CHAC1/BOTCH by the androgen receptor (AR) and Jumonji-domain containing protein-3 (JMJD3) led to elevated NOTCH1 levels and exacerbated neuroinflammation ( ATF4 was most prominently upregulated, though other related genes were also significantly elevated (Yasuda et al., 2014)

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However, excessive H 2 O 2 accumulation can lead to oxidative damage to proteins, lipids, and nucleic acids, highlighting the need for tight regulation by catalase and other antioxidant systems

Similar studies also suggested that such NEPPs from waste products have a remarkable antioxidant content, resultantly may exhibit health promoting effects in the cornerstone of the human diseases prevention (557)

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