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[DOI] [PMC free article] [PubMed] [Google Scholar] 30.Huang T, Zhang K, Sun L, Xue X, Zhang C, Shu Z, et al

Effects of the macular carotenoid lutein in human retinal pigment epithelial cells

Hyperactivation of oncogenes leads to excessive replication stress and activation of the DDR pathway.15 Oxidative Stress and Mitochondrial Dysfunction: Reactive oxygen species (ROS) generated from mitochondrial dysfunction or environmental stress contribute to cellular senescence.16 Oxidative stress leads to DNA damage, lipid peroxidation, and protein dysfunction.17 ROS can activate the p38 MAPK pathway, which enhances p53 and p16 signaling, reinforcing senescence.18 Importantly, oxidative stress is not an isolated mechanism but a central upstream driver of the SASP

& Glass, C

doi: 10.1016/j.cmet.2020.06.010

Both direct and indirect effects account for the pro-inflammatory activity of enteropathogenic mycotoxins on the human intestinal epithelium: stimulation of interleukin-8 secretion, potentiation of interleukin-1 effect and increase in the transepithelial passage of commensal bacteria