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This evidence leads us to hypothesize that, in the cortical motor areas of SMA mice, dysregulations in PV + IN GABA synthesis trigger a disruption of inhibitory neurotransmission in the late disease stage, which occurs simultaneously and in response to late-stage impairment of Gln neuron-astrocyte transport and/or metabolism
The adaptive, evolutionary significance of disrupted synaptic plasticity in response to stress is not clear
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Endothelial senescence and chronic fatigue syndrome, a COVID-19 based hypothesis
2021 Apr;28(2):349-359
Acknowledgements The images were created with Biorender software
