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This phenomenon may be attributed to the formation of the homodimer

Interleukin-4 (IL-4) and 12/15 lipoxygenase (12/15-LOX) contribute to mitochondrial dysfunction in allergic asthma and can be reduced by vitamin E supplementation (Mabalirajan et al., 2009)

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However, in the past decade, multiple lines of scientific evidence challenged this traditional dichotomy

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