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Therefore, a high level of cellular GSH may still partly feedback-inhibit even if the GSH1 is under the control of a constitutive promoter pTEF1 , i.e., by inhibiting Gsh1 enzyme activity

This GSH conjugation is spontaneous or enzymatic via GSH-S-transferases [21,22]

[DOI] [PubMed] [Google Scholar] 155.Okunade O., Niranjan K., Ghawi S.K., Kuhnle G., Methven L

Clinical activity of pazopanib in patients with advanced desmoplastic small round cell tumor

Protein tyrosine nitration of the flavin subunit is associated with oxidative modification of mitochondrial complex II in the post-ischemic myocardium

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