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HER2 confers resistance to foretinib inhibition of MET-amplified esophageal adenocarcinoma cells

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However, emerging research suggests that similar pathways may operate in mammals, where mitochondrial toxicants or agents that alter the redox environment could have heritable effects on offspring development and health

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To support this demand, HCC cells avidly take up exogenous glutamine through transporters such as ASCT2 and SNAT1 (SLC1A5/SLC38A1) [72,73]

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