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Description
Clin Cancer Res 1997
Redoxpotential
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Given that KRAS mutations are prevalent in almost 30% of non-small cell lung cancer (NSCLC) patients [2], we chose a Kras G12D -driven spontaneous lung cancer murine model to elucidate the detailed mechanism by which DDX3X regulates lung cancer progression, which might be also extended to KRAS wildtype lung cancers
b: The major mechanism of release involves a cytosolic phospholipase A 2 (cPLA 2 )
Molecular mechanisms of cisplatin resistance in cervical cancer
