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ROS is widely acknowledged as an upstream mediator that promotes the activation of the NLRP3 inflammasome, triggering subsequent inflammatory cascades (66)
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These findings collectively suggest that Aurora-A kinase, even without T-loop-phosphorylation, can phosphorylate certain substrates like Lats2 and TACC3, most likely in a complex with its co-activator TPX2
Elmasta et al
More recently, the U.S
