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This again suggests the critical role of peroxynitriterather than LPOas the primary cause of oxidative damage to mitochondrial DNA 18 and protein nitration, which are central mechanisms of toxicity (Fig

A subsequent study revealed that CoCl 2 pretreatment protected auditory hair cells (HEI-OC1) from H 2 O 2 -mediated cytotoxicity via the activation of the redox-sensitive transcription factors HIF-1 and NRF2, as well as their target gene PRDX6 , indicating the protective roles of antioxidant enzymes against noise-triggered oxidative damage [136]

[DOI] [PMC free article] [PubMed] [Google Scholar] 115.Chan K, Han XD, Kan YW

Neurosci Lett 741:135504

Pulmonary defense is dependent on the immune and nervous systems

Folate usually exists in the body as tetrahydrofolate (THF), which has extra glutamate residues, making it a polyglutamate