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Our findings revealed that chemogenetic deactivation of DMH VGAT neurons decreased the number of DNA fragmented cells indicative of reduced reperfusion injury (Fig
doi:10.1007/s11010-021-04106-4 Le Sayec M, Xu Y, Laiola M, et al
p=0.04)
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This CEBPBHIF-1GLUT1 axis highlights CEBPBs role in metabolic adaptation to hypoxia, facilitating metastatic progression
doi: 10.1093/ajcn/81.1.95
