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Synaptic stimulation via the action of glutamate and D-Ser on ion channels activates downstream signaling and promotes protein synthesis, which is required for the long-term synaptic response and synaptic remodeling

Active cutaneous infections (bacterial, viral, or fungal)

Azide inhibition of mitochondrial electron transport

as a result, tolerance towards the commensal intestinal microbiota is lost, causing tissue damage and chronic inflammation

In lung cancer, the TME favors M2 polarization, characterized by enhanced OXPHOS, FAO, and upregulation of ARG1 and IL-10

When CREB is phosphorylated by JNK, its ability to activate the BDNF promoter is diminished, resulting in decreased BDNF production (Motaghinejad et al