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Thus, there is consistent and quite complete evidence (i.e., reduction in all of the routine necrosis markers, grossly no visible infarcted area, attenuated histological damage, ECG (no ST-T ischemic changes), and echocardiography (preservation of systolic left ventricular function) damage and oxidative stress parameters decreased) [38]

Clin Imaging 37(4):631636 Do Van B, Gouel F, Jonneaux A, Timmerman K, Gel P, Ptrault M et al (2016) Ferroptosis, a newly characterized form of cell death in Parkinsons disease that is regulated by PKC

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Although energy metabolism status can also regulate intracellular ROS levels (e.g., glucose deprivation leads to a marked increase in cellular ROS) [53,54,55,56], our study demonstrated that supplementation with energy substrates such as sodium pyruvate in GGCT-knockdown cell models did not induce significant alterations in intracellular ROS levels

or use of antitumor necrosis factor (TNF)- or EPO or treatment with immunoglobulin drugs 6 months before or during ARA 290 administration and in the follow-up phase

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