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Ginsenoside Re can effectively prevent METH-induced mitochondrial dysfunction, oxidative damage, microglial activation, activation of proapoptotic factors, and degeneration of dopaminergic neurons by inhibiting the PKC gene (Shin et al., 2014
That said, the safety record is excellent with minimal side effects
Stimulating growth factor
2016;5:e003682
Surgery such as joint replacement or joint fusion may be recommended to remove tophi or manage joint damage
