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(2003) found that hypothyroidism caused oxidative stress in the liver and heart, causing an imbalance between ROS and endogenous antioxidant body defenses, with subsequent damage to cellular macromolecules ( L-thyroxine administration moderated such oxidative stress in hypothyroid rats through normalizing the plasma MDA level and plasma TAC, in agreement with Varghese et al

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Metabolites like lactic acid can lactylate histones, promoting pro-fibrotic gene expression in lung macrophages, contributing to radiation-induced pulmonary fibrosis (RIPF) [14]

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Taken together, these factors support a mostly positive trust assessment, although routine verification is still reasonable

P = 0.0016