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This finding may be compatible with the previous reports indicating that ERGO modulates oxidative damage of the kidney in rats and that decreased ERGO levels may contribute to chronic kidney disease progression implying beneficial roles of ERGO in the kidney [80, 81]

Ann N Y Acad Sci 2004;1033:158-67

Mitochondrial dysfunction in pathophysiology of heart failure

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In sum, the evidence strongly supports membrane perturbation and altered phospholipid packing in mediating the electrophysiological and inotropic effects of LCACs in the heart

73 McCoinC